Diagnostic
Summary: Disease and Adaptive Models of Addiction Impact on DSM-5-TR
Diagnoses
M. Aly’ce Wilson
College of Allied Health, Walden
University
CPSY-8781-2: Psychopathology from a
Clinical Perspective
Dr. Megan Corley
May 4, 2025
Diagnostic
Summary: Disease and Adaptive Models of Addiction Impact on DSM-5-TR
Diagnoses
Introduction
Understanding the psychopathology of diseases
including the homotypic and heterotypic continuity is foundational. Diagnostic process’ includes person-centered symbiotic
holistic assessments for competently and effectively treating individuals. The DSM-5-TR
Part One: Diagnostic
Summary
Diagnostic
Impression: Opioid Use Disorder DSM 304.00/(F11.20) ICD-11 Moderate
Jane
Doe states her main concern for current presentation is her “chronic pain
official pain syndrome.” Jane attributes
her pain in neck and shoulders that “lingers for days” to affecting her sleeping. There are times the pain is so bad Jane cannot
“get out of bed in the morning.” Jane’s
appearance is well groomed, appropriate, clean, and hygienic. Jane’s eye-contact was fitting, normative
gate and a minor slouching posture. Jane’s
orientation indicates a decline in cognitive functioning as evidence by her
statements of, “I'm sorry, because you're making me confused with your
questions… I don't know. I'm sorry. What was… What
did you ask?” and that she is “fucking terrible with names.” Jane’s thought process is indicative of once
having the ability to be goal-directed/logical, however her inability to answer
questions without giving excessive and unnecessary details is
circumstantial. Observation of Jane’s irritable
affect and oppositional behavior intensified when presented with evidence that
did not align with her goal achievement as evidence by her tone, body language,
and impatience towards an office assistant.
Jane’s thought content appears to be distorted with the preoccupation of
obtaining “oxycodone” for her myofascial pain manifestations. Jane states she ran out of her (15mg)
Oxycodone prescription from Dr. Wade, her primary care physician (PCP) a week
before it was due to be filled; the PCP refilled that prescription with another
15 pills that had been consumed before her next refill was due to be filled as
well. Jane’s PCP lowered the dosage for Jane
calling it a “trial.” Jane states “the
(50mg) dose of OxyContin is really the only dose that helps” indicating the
need for increased amounts with diminished effect as evidence by the high
tolerance. Jane admits to having two
additional prescriptions, but did not indicate the dosage. Jane has a history of “trying to get all the
way off” only because she ran out of the pharmaceutical as well as having “to
call out for work.” Jane’s having “seen
5 doctors in 2 days” indicates a considerable amount of time dedicated toward
med seeking behavior and away from occupational responsibilities. Jane has not completed the physical therapy
that she was referred for pain management due to her “not liking the guy.” Jane denies interest in finding another physical
therapist or alternatives such as acupuncture, except for massage therapy that
she does not have insurance coverage to participate. Jane states she “might” take Oxycodone on
days she does not have pain. Jane states
she “sometimes crushes or chews” her Oxycodone.
Jane acknowledges side effects from Oxycodone such as “dizziness.” Jane denies the use of any other substances
except for “sometimes” alcohol consumption and in high school where she “piggy-backed
off her friends Adderall.” Jane denies
prior drug or alcohol arrests. Jane
denies prior inpatient or outpatient treatment for drugs or alcohol.
Jane
meets the provisional diagnosis of opioid use disorder with moderate severity
that without intervention homotypic continuity will worsen the severity and impact
her life functioning to a greater degree.
Further evaluation is needed to assess for and rule out if opioid withdrawal
diagnosis and treatment is appropriate.
There is no factual knowledge at this time of Jane’s last acquisition of
Oxycodone. However, her presence in the fifth
doctor’s office in two days is an indication of cessation with symptomatic
dysphoric mood not having been able to obtain another prescription, muscle
aches, and insomnia causing significant pragmatic deficits in life functioning
attributed to missing work. While Jane
currently denies excessive stress and anxiety, continuing evaluation is needed
to assess heterotypic continuity as life functioning deficits typically continue
to worsen without intervention.
Part Two: Disease and
Adaptive Models of Addiction Impact DSM-5-TR Diagnosis
Addiction
is complex with many subtypes. The
symbiotic effects emergent through symptomatic behavioral expression in individuals
can be reversely traced when a baseline agreement becomes attainable, “substance
abuse disorders do not develop in isolation”
Disease Model
Jellinek (1960) promoted a “new approach” regarding the disease
concept of alcoholism theory. The
disease model closely aligns with the medical model Dr. Silkworth (1939) pioneerd
withis his application of alchol dependence
Advantage
Evolutionary advancement
toward a societal problem that has been deemed a moral turpitude for millennia,
being found erroneous scientifically has given way to social reform on a global
scale. Research and application on homotypic
and heterotypic continuity of disease intertwining with substance-related and
addictive disorders has transformed society.
Continued research finding causation has influenced the beginning of protections
for children, women and other marginalized and otherwise under-represented
minorities such as racial over-incarceration.
Social support has been made available on a socioeconomic level that was
previously available only to the wealthy, veterans, or men.
Drawback
Allowing behaviors
associated with addiction to be classified as a disease begs the hypocritical dilemma
on moral deficiency of those who claim empathy for the sick as virtuous. This pits the cultural domains of religion
and government who have tried to eradicate addiction against scientific and
academic communities. Cultural evolution
is a slow-moving process. The balance
between responsibility for one’s own actions and the inability to control them place
this at the center of debate. For
example: Individuals with a physical disease diagnosis of lung cancer if
the cause is socially unacceptable behavior and preventable such as smoking are
arguably undistinguishable within a society for whom it affects .049% of which
10-20% having never smoked
DSM-5-TR Impact
The first DSM published by the APA (1952) identified drug addiction and
alcoholism catagorically with sociopathic personality disturbance although
there were not any diagnostic systems ensuring relability or validity
Adaptive Model
Alexander and Hadaway (1982) introduce an adaptive
orientation model in opiate addiction. Alexander
(1987) postulates addiction is a substitute for failing to “reach or maintain
adult integration” ultimately leading to suicide
Advantage
The adaptive model
recognizes and introduces a new theory of cause-and-effect in that addiction is
not the cause of the pragmatic deficits of function, but a solution
for the individual despite the consequences of maladaptive behavior. Alexander (1987) opines there is no addict
per se, there is a mere societal change that now demonizes drugs and alcohol
where before when it was more socially acceptable there were less individuals with
functional deficits. Society he discoursed
should be accountable for the problems it faces rather than look for a
scapegoat. “Large numbers of people
suspected of distributing drugs have been intimidated, deprived of their jobs,
property, crops, and civil rights, and subjected to long prison terms”
Drawback
The adaptive model fails
to consider early onset adolescent addiction.
The adaptive model opines a “search and choice” as if anyone in the US
need “search” for alcohol, tobacco, or caffeine sold at every corner store as a
“convenience.” Alexander (1987) fails to
recognize obsessive compulsive urges and craving that can occur without
prolonged exposure. His arguments of
either/or with several sub-associations including: behavior, treatment, and environment,
etc. fail to recognize two opposing truths coexisting simultaneously. The introduction of polarizing models of
addiction while both beneficial and lacking in some asspects contributed to the
evolution of the DSM-III at the time, into the DSM-IV and current DSM-V-TR, respectively.
DSM-5-TR Impact
The DSM-IV focused heavily on functional deficits for the individual
including criminal activity. The DSM-IV nuanced
“abuse” from “dependence” as well as adding severity specifiers such as mild,
moderate, and severe (APA, 2000). The
current DSM-5-TR removed judicial consequences focusing instead on differentiating
intoxication and withdrawl. Criteria including:
nuanced differences with nonpathaological use, there are comorbidity
acknowledgments, functional consequence applications, advances in clinical and
emperical evidence with the symbiotic nature of human development, personality,
and sociological and economical cobtributions to risk and protective factors,
as well as development and course beyond the gentic, bio, and neurological
predispositions. While evolution is slower
than the metaphorical wheels of justice, social reform prompted by the disease
model of addiction has undoubably ushered in chang. The infulenctail impacts to the DSM-5-TR began
with a theory and developed objectifiable drawbacks and advantages.
Conclusion
Standard diagnostic processes, assessments,
models, foundational understanding of homotypic and heterotypic continuity in psychopathology
of disease, human development, personality, social, cultural sensitivity, awareness
of bias’, as well as objectively and critically analyze discourse in research
are applied holistically, individually for those who are experiencing pragmatic
functional deficits, for which they are seeking help to mitigate as a person, symbiotically
experiencing an expansive-small world.
References
Alcoholics Anonymous. (2002). Alcoholics
Anonymous (4th ed.). New York, NY: Alcoholics Anonymous World Services.
Alexander, B. K. (1987). The disease and adaptive
models of addiction: A framework evalution. The Journal of Drug Issues, 17(1),
47-66. Retrieved from DOI:10.1177/002204268701700104
Alexander, B. K., & Hadaway, P. F. (1982).
Opiate addiction: The case for an adaptive orientation. Psychological
Bulletin, 92(2), 367-381. Retrieved from 0033-2909/82/9202-0367S00.75
American Psychiatric Association. (1952). Diagnostic
and Statistical Manual: Mental Disorders. Washington, DC, USA: American
Psyciatric Association.
American Psychiatric Association. (1968). Diagnostic
and statistical manual of mental disorders (2nd ed.). Washington,
Washington, DC, USA: American Psychiatric Association.
American Psychiatric Association. (1980). Diagnostic
and statistical manual of mental disorders (3rd ed.). Washington, DC,
USA: American Psychiatric Association.
American Psychiatric Association. (2000). Diagnostic
and statistical manual of mental disorders (4th ed.). Washington, DC, US:
American Psyciatric Association.
American Psychiatric Association. (2022). Diagnostic
and Statistical Manual of Mental Disorders, Fifth Edition, Text Revision.
Washington, DC: American Psychiatric Association.
American Psychological Association. (2017). Ethical
principles of psychologists and code of conduct. American Psychological
Association. Retrieved from https://www.apa.org/ethics/code
Hunt, A. (2015). Expanding the biopsychosocial
model: The active reinforcement model of addiction. Graduate Student
Journal of Psychology, 15, 1-13.
Jellinek, E. M. (1960). The new approach. In The
disease concept of alcoholism. New Haven, CT: Hillhouse Press. Retrieved
from doi:10.1037/14090-001
Jung, C. G. (1963). The Bill W. - Carl Jung letters.
Retrieved from
https://www.silkworth.net/wp-content/uploads/2020/07/The-Bill-W-Carl-Jung-Letters-Jan-1963.pdf
National Cancer Institute. (2024). U.S. Cancer
statistics working group. U.S. cancer statistics data visualizations tool. .
Centers for Disease Control and Prevention, U.S. Department of Health and
Human Services. Retrieved from https://www.cdc.gov/cancer/dataviz
Norko, M. A., & Fitch, W. L. (2014). DSM-5 and
substance use diorders: Clinicolegal implications. American Academy of
Psychiatry and the Law, 42(4), 443-452. Retrieved from
https://jaapl.org/content/42/4/443
Somnath, P. (2018). Incidence and prevalence of
major neurologic disorders. US Pharm, 43(1), 24. Retrieved from
https://www.uspharmacist.com/article/incidence-and-prevalence-of-major-neurologic-disorders
Substance Abuse and Mental Health Services
Administration. (2012). Brief Interventions and Brief Therapies For
Substance Abuse (Vol. TIP 34). Rockville, MD, USA: U.S. Department of
Health and Human Services. Retrieved from
https://store.samhsa.gov/sites/default/files/d7/priv/sma12-3952.pdf
Substance Abuse and Mental Health Services
Administration. (2023). Highlights for the 2023 national survey on drug
use and health. Retrieved from
https://www.samhsa.gov/data/sites/default/files/NSDUH%202023%20Annual%20Release/2023-nsduh-main-highlights.pdf
Suris, A., Holliday, R., & North, C. S. (2016).
The evolution of the classification of psychiatric disorders. Behavioral
Sciences, 6(1), 5. Retrieved from https://doi.org/10.3390/bs6010005
Symptom Media (Producer). (2018). Training Title
143 [Motion Picture]. Retrieved from
https://video.alexanderstreet.com/wayf?account_id=14872&redirect=%2Fwatch%2Ftraining-title-143
Tsermpini, E. E., Goričar, K., Plesničar, B.,
Plemenitaš Ilješ, A., & Dolžan, V. (2024). The disease model of
addiction: The impact of genetic variability in the oxidative stress and
inflammation pathways on alcohol dependance and comorbid psychosymptomatology.
Antioxidants, 1-20. Retrieved from
https://doi.org/10.3390/antiox13010020
World Health Organization. (2019). International
statistical classification of diseases and related health problems (11th Ed).
Comments
Post a Comment